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UCLan Year 4 BDS: Dental Anomalies · Meedu
Course
Dentistry
UCLan Year 4 BDS: Dental Anomalies
Dental Anomalies 1
Dental Anomalies 2
Dental Anomalies 3
Amelogenesis Imperfecta
Molar Incisor Hypomineralisation
UCLan Year 4 BDS: Dental Anomalies
Year 4 BDS Dental Anomalies module rebuilt to the UCLan syllabus. Three lectures covering the spectrum of developmental and acquired anomalies of the dentition, plus core reading on amelogenesis imperfecta and molar incisor hypomineralisation.
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Which mechanism best explains why supernumerary teeth in cleidocranial dysostosis typically exhibit normal morphology compared to isolated mesiodens?
Select the correct answer
A.
Cleidocranial dysostosis involves continued dental lamina activity producing organised tooth formation, whilst isolated supernumeraries result from focal hyperactivity causing dysmorphic development
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B.
The genetic mutation in cleidocranial dysostosis prevents enamel organ malformation, whereas sporadic supernumeraries develop from aberrant epithelial remnants
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C.
Systemic conditions produce supernumeraries during normal odontogenesis timing, whilst isolated cases arise from delayed ameloblast differentiation
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D.
Cleidocranial dysostosis supernumeraries develop from primary dental lamina, whereas isolated mesiodens originate from successional lamina tissue
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E.
The RUNX2 gene defect in cleidocranial dysostosis accelerates mineralisation, producing well-formed teeth compared to sporadic developmental arrests
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